P02-002 - IL36RN mutations in patients with DITRA

نویسندگان

  • JI Arostegui
  • MA Vicente-Villa
  • A Chaves
  • E Gonzalez-Roca
  • E Ruiz-Ortiz
  • J Rius
  • S Plaza
  • MA Gonzalez-Enseñat
  • J Yague
چکیده

Introduction Loss-of-function mutations in the IL36RN gene define a novel recessively inherited autoinflammatory syndrome named deficiency of IL-36 receptor antagonist (DITRA). This genetically determined deficiency was first described in a subgroup of patients with generalized pustular psoriasis. It is a life-threatening condition characterized by recurrent episodes of severe skin inflammation, with pustule development, associated with fever, malaise, extracutaneous involvement, neutrophilia and a marked acute phase response.

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Generalized pustular psoriasis in infant with heterozygous mutation in the IL36RN gene successfully treated with infliximab

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Introduction Interleukin-36-receptor antagonist deficiency (DITRA) is a recently described auto-inflammatory disease, characterized by repeated flares of generalized pustular psoriasis, high fever, asthenia and systemic inflammation. This condition is caused by homozygous missense mutation in the IL36RN gene, encoding the interleukin-36-receptor antagonist (IL-36Ra), an anti-inflammatory cytoki...

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عنوان ژورنال:

دوره 11  شماره 

صفحات  -

تاریخ انتشار 2013